By David Kritchevsky, Kenneth K. Carroll

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If one stops ingesting ω-3 fatty acids, then, any beneficial effect would ultimately be lost as these fatty acids are slowly catabolized. • Populations to which scientific evidence can be generalized: Epidemiologic studies performed in a number of countries illustrate an association between ω-3 fatty acid intake and the beneficial effects discussed above. These countries and/or population subsets would include the following: Greenland Eskimos; Alaskan natives; Japanese farmers; and hyperlipidemic, hypertensive, and coronary patients in the United States, Germany, Scandinavia and Great Britain.

National Academy Press. p. 8,671. National Research Council, Food and Nutrition Board. 1989b. Recommended dietary allowances. 10th. ed. Washington, DC: National Academy Press. p. 48. J. 1986. Fish oil attenuates the cholesterol induced rise in lipoprotein cholesterol. Am. J. Clin. Nutr. 43:752–757. P. 1991. Atherosclerosis in Alaskan natives. FASEB J. 5:A1252. (Abstract). ; Feychting, M. 1986. Fish consumption and mortality from coronary heart disease. Br. Med. J. 293:426. J. 1986. Effect of dietary supplementation with fish oil on systolic blood pressure in mild essential hypertension.

1991. Inverse association between fish intake and risk of glucose intolerance in normoglycemic elderly men and women. Diabetes Care 14:935–941. ; de W. S. 1991. Atherosclerosis: chronic effects of fish oil and a therapeutic diet in nonhuman primates. Arterioscler. Thromb. 11:719–732. ; Osland, A. 1990. Fishoil concentrate: effects on variables related to cardiovascular disease. Am. J. Clin. Nutr. 52:300–306. E. 1988. Fish oils inhibit endothelial cell production of platelet-derived growth factor-like protein.

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