By L. Rea
After all, being during this company for 15+ years myself ... and working with pretty well all of the significant humans and authorities (Dr. Scott Connely, Joe Weider, Dan Duchaine, etc.) I had my very own doubts. in reality, i began this particular enterprise as a studying scan through hiring ALR as my very own own trainer simply to see if he was once ''for real''.After talking to him for plenty of hours and days, I speedy got here to gain simply how clever and an expert he was once approximately this complete ''chemistry'' of the physique. Please word, that I acquired into clinical college myself ... so i am no newbie in terms of these items.
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200 mg, Day #1 9-20 Deca 1 50 mg/Primo D. 250 mg. 48 Example #1A has been adjusted to accommodate an intermediate athlete's needs by either cutting dosages in half or by utilizing the injections scheduled on days#1,3,5,7,9,20,22,24. Example #1B was adjusted for the same athletes simply by cutting listed dosages in half. 5 mg/D. "... " Each of these examples allowed Frank and other beasts to get in, grow hard and get out before the body could mount a full growth hating counter attack. With that said, let's go to the next page and check out Example #2, chart and description 49 MAX ANDROGEN PHASE Example #2 - Chart DAY 1.
Normally the body utilizes cholesterol to synthesize sex hormones endogenously (produced inside the body). Testosterone, estrogen and others all begin as cholesterol. When AAS are introduced exogenously, this process is inhibited. This in turn allows cholesterol to build-up to dangerous levels until discontinuance of the AAS. An interesting fact is that anything that stimulates HPTA (hypothalamuspituitary-testes-axis) function/activity also decreases bad cholesterol levels. It does so by increasing hormone (like testosterone) production endogenously.
Okay, we know the body adapts by reaction to AAS beginning after 2-3 weeks. We know that some AAS aromatize to estrogen which needs to be checked and eliminated before we allow you to exit your AAS protocol, Frank. But we also know estrogen levels can actually enhance AAS results by several pathways including increased GH/IGF-1 production and increased muscle glycogen synthesis. We also know any androgenic induced muscle mass gains not solidified into high quality lean muscle tissue by a high anabolic environment will be lost quickly post-cycle.